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THE MOLECULAR-BASIS OF GENETIC DOMINANCE
Author(s): WILKIE AOM
Source: JOURNAL OF MEDICAL GENETICS    Volume: 31    Issue: 2    Pages: 89-98    Published: FEB 1994  
Times Cited: 117     References: 124     
Abstract: Studies of mutagenesis in many organisms indicate that the majority (over 90%) of mutations are recessive to wild type. If recessiveness represents the 'default' state, what are the distinguishing features that make a minority of mutations give rise to dominant or semidominant characters? This review draws on the rapid expansion in knowledge of molecular and cellular biology to classify the molecular mechanisms of dominant mutation. The categories discussed include (1) reduced gene dosage, expression, or protein activity (haploinsufficiency); (2) increased gene dosage; (3) ectopic or temporally altered mRNA expression; (4) increased or constitutive protein activity; (5) dominant negative effects; (6) altered structural proteins; (7) toxic protein alterations; and (8) new protein functions. This provides a framework for understanding the basis of dominant genetic phenomena in humans and other organisms.
Document Type: Review
Language: English
Addresses:
1. UNIV HOSP WALES, INST MED GENET, CARDIFF CF4 4XW, WALES
Publisher: BRITISH MED JOURNAL PUBL GROUP, BRITISH MED ASSOC HOUSE, TAVISTOCK SQUARE, LONDON, ENGLAND WC1H 9JR
Subject Category: Genetics & Heredity
IDS Number: MW006
ISSN: 0022-2593
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