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EVIDENCE FOR DEGRADATION OF MESSENGER-RNA ENCODING ALPHA-L-IDURONIDASE IN HURLER FIBROBLAST WITH PREMATURE TERMINATION ALLELES
Author(s): MENON KP, NEUFELD EF
Source: CELLULAR AND MOLECULAR BIOLOGY    Volume: 40    Issue: 7    Pages: 999-1005    Published: NOV 1994  
Times Cited: 29     References: 39     
Abstract: Mutations in the gene encoding alpha-L-iduronidase (IDUA) are the cause of Hurler syndrome. Fibroblasts from patients homozygous for nonsense IDUA alleles have much reduced mRNA detectable by Northern analysis, as has been observed in many other instances of premature translation termination. Yet RT-PCR (reverse transcription followed by PCR amplification) showed a normal level of a segment covering exons 1 and 2 in Hurler cells homozygous for alleles bearing the nonsense mutations, Q70X or W402X. The 3' end of the segment was between exons 2 and 4. The results indicate that the nonsense RNA was degraded to fragment(s), independent of the position of the mutation (exon 2 or exon 9, respectively). Treatment of the cells with cycloheximide resulted in some increase of intact mRNA, suggesting that translation is required for mRNA degradation.
Document Type: Article
Language: English
Addresses:
1. UNIV CALIF LOS ANGELES, SCH MED, DEPT BIOL CHEM, LOS ANGELES, CA 90024 USA
Publisher: CELLULAR & MOLECULAR BIOLOGY, PROF R WEGMANN RESIDENCE HAUSSMANN 1 AVENUE DU PAVE NEUF, 93160 NOISY-LE-GRAND, FRANCE
Subject Category: Biochemistry & Molecular Biology; Cell Biology
IDS Number: PN628
ISSN: 0145-5680
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